Kyphoscoliosis Ehlers-Danlos (formerly called EDS type VI)

 

 

Characteristics. It is the only EDS with Autonomic Recessive Inheritance and is characterized because the newborn can have severe muscular weakness and scoliosis or develop it during the first year of life. They also present joint hypermobility, recurrent sub-luxations, and friable, lax skin, with frequent ecchymoses and with ocular problems, consisting in fragility of the sclera, with occasional rupture of the ocular globe. Some may have marfanoid habitus. Another possible complications are rupture of medium size arteries and these children may have poor development of muscular tone. One hundred percent of them have osteoporosis. It is a rare disease, seen in 1 out of 100,000 born alive children.

 

Since it has Autonomic Recessive Inheritance, both parents are heterozygotes. The altered gene is the PLOD that is localized at 1p36.3-p36.2 and is a protein, the Procollagen-Lysine, 2 Oxoglutarate, 5 Dioxygenase. The parents (heterozygotes), that are asymptomatic, have only one copy of the altered gene PLOD that causes the illness.  Siblings have 25% chances to get the illness, 50% chances to be asymptomatic carriers and only 25% chances to be healthy and not carriers. The children of patients with Kyphoscoliosis EDS are heterozygotes and their children have 50% chances to be also heterozygotes.

 

Diagnostic criteria for the Kyphoscoliosis EDS.

 

A.-  Mayor signs:

 

-          Friable, thin, hiperextensible skin with frequent ecchymoses.

-          Generalized joint hypermobility.

-          Severe muscle weakness, as a newborn.

-          Progressive scoliosis present at birth or during the first year of life.

-          Fragility of the sclerae or rupture pf the ocular globe.

 

B.-  Minor signs:

 

-          Atrophic or wide surgical scars.

-          Marfanoid habitus.

-          Medium size artery rupture.

-          Mild or moderate delay in achieving the normal motor strength.

 

For making the diagnosis.  The presence of 3 mayor signs is highly suggestive of the diagnosis of Kyphoscoliosis EDS.

 

Diagnostic confirmation.  Increase value of the ratio between the urinary deoxypiridinoline and piridinoline, measured by the HPLC (a very sensitive and specific test). The genetic confirmation is possible only as a research tool and not available in private practice.

 


Treatment.

 

Of the musculo-skeletal problem:

 

-         Orthopedist referral for treatment of the Kyphoscoliosis, which at times requires surgical correction.

-         Physiotherapy referral to increase strength of the big muscles, like the shoulder girdle, to prevent recurrent shoulder subluxations.

 

Of the cardiovascular problem:

 

-         Measure the aortic root dilatation with an ECO., starting at age 5 and repeating the test every 5 years if the test is negative. Yearly when dilation exists.

-         Prophylactic treatment of the mitral valve prolapse.

-         Careful observation of the blood pressure, reducing it in order to prevent arterial ruptures (Losartan). Vascular surgery is dangerous, due to arterial wall fragility.

 

Of the ocular problem:

 

Early detection of Myopia and Glaucoma are important.

 

Of others problems:

 

-          Early detection of inguinal hernias, for surgical treatment.

-          The use of vitamin C can increase hydroxilysine excretion and improve muscle strength, it also improves scar cicatrisation. Doses are from 500 mg a day in children up to 10 gm a day in adults.

-         Good hydration is needed to prevent oxalic nephrolitiasis.

 

 

 

Jaime F. Bravo, MD.

Rheumatology – Osteoporosis

Revised: July 12th, 2007

 

 

 

Dr. Bravo’s Rheumatology web site